can anyone prove it doesnt?
I can put forward a pretty good argument that it doesnt, but as i said it is long winded and technical. happy to do so when i have the time. I used to have a website that explained it all, but it was hacked and destroyed.
Lets be serious it cant be good for the ticker can it.
care to explain why not?
All i did was stop smoking.
thats all you did? you did one of the most beneficial things you can do for your health. Considering the repair role that cholesterol plays in your body, it is only to be expected that your cholesterol will drop once you stop poisoning it.
Its like saying that once i stopped cutting myself i stopped getting scars
The repairing cholesterol is high density whereas the build up from smoking is low, so your explanation is a bit off..
actually there is only one type of cholesterol, and that is cholesterol.
High density and low density actually refers to Lipoproteins. HDL is a high densty lipoprotein, etc. There are also Intermediate Lipoproteins and Very Low Density Lipoproteins. You see, this is where the smoke and mirrors starts, HDL and LDL are not actually cholesterol, they are the things that transport cholesterol (and fat)around your body.
In a nutshell they start of as Very Low Density lipoproteins and they transport Cholesterol and fat around the body and deliver them where they are needed. this is done only be means of specific receptors. Fat or cholesterol cannot be dumped from lipoproteins willy nilly, and if they were, they would not even stick to the endothelium, the "non stick" inner lining of our arteries.
a VLDL will have mainly tryglycerides (fatty acids or fat in simple terms) and cholesterol. the fatty acids will be delivered to cells that require energy, or stored as fat in the body.
as it loses these fatty acids it becomes an Intermediate density lipoprotein.
at this stage it can actually be reabsorbed by the liver and i believe this to be crucial, but noone seems to know what determines whether it is reabsorbed, or whether it continues on its delivery run.
as it continues to loose the tryglycerides (and the "key" that would have allowed it to be reabsorbed by the liver) it now becomes an LDL, , containing mainly cholesterol and some fat. It's purpose is to deliver cholesterol and fat soluble vitamins to cells that require it.
A HDL is a different cat. it circulates around exchanging "keys" that lock into receptors with the VLDs/IDLs/LDLs , as well as scavenging used cholesterol from cell walls and returning it to the liver to be reprocessed.
Now for cholesterol to end up in our arteries, we first need something to damage the endothelium, the body then starts a repair process. This is when so called arterial plaque is formed.These arterial plaques are basically scar tissue. Of the eight stages of this plaque formation, cholesterol and fat appear at stage 6 (now that hardly be the cause of the problem, can it?).
One thing known to damage the endothelium is nicotine. So when Daniel stopped smoking, he reduced the amount of damage done to his arteries, amongst other things. He therefore reduced his body's need for cholesterol, so his LDL dropped accordingly. (this would be due to reduced damage to cells all through the body, not just in the arteries.)